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Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells [RNA-Seq]

ABSTRACT: Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells [RNA-Seq]

PROVIDER: PRJNA847204 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR19581700_1.fastq.gz	Fastqsanger.gz
	SRR19581700_2.fastq.gz	Fastqsanger.gz
	SRR19581701_1.fastq.gz	Fastqsanger.gz
	SRR19581701_2.fastq.gz	Fastqsanger.gz
	SRR19581702_1.fastq.gz	Fastqsanger.gz

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Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells

Project description:Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells

| PRJNA847198 | ENA

Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells [miRNA-Seq]

Project description:To investigate the effect of gene alteration of Miat and Mtdh in mouse stem-like MB cells. by canonical oncogenic signaling to maintains the self-renewal capacity of a population of tumorigenic stem-like medulloblastoma cells that are resistant to cytotoxic therapy. by canonical oncogenic signaling to maintains the self-renewal capacity of a population of tumorigenic stem-like medulloblastoma cells that are resistant to cytotoxic therapy. Miat and Mtdh cooperate to promote medulloblastoma tumorigenesis and treatment resistance by regulating the biogenesis of a subset of microRNAs that target genes involved in these processes.

2022-07-29 | GSE205694 | GEO

Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells [RNA-Seq]

Project description:To investigate the effect of gene alteration of Miat in mouse stem-like MB cells. by canonical oncogenic signaling to maintains the self-renewal capacity of a population of tumorigenic stem-like medulloblastoma cells that are resistant to cytotoxic therapy. by canonical oncogenic signaling to maintains the self-renewal capacity of a population of tumorigenic stem-like medulloblastoma cells that are resistant to cytotoxic therapy. Miat and Mtdh cooperate to promote medulloblastoma tumorigenesis and treatment resistance by regulating the biogenesis of a subset of microRNAs that target genes involved in these processes.

2022-07-29 | GSE205691 | GEO

Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells [miRNA-Seq]

Project description:Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells [miRNA-Seq]

| PRJNA847203 | ENA

Gene expression of depletion of lncRNA Miat in mouse stem-like MB cells

Project description:This SuperSeries is composed of the SubSeries listed below.

2022-07-29 | GSE205695 | GEO

Transcriptome profiling of left ventricles from systemic MIAT knockout mice

Project description:Genome-wide transcriptomic analyses in left ventricles (LVs) from systemic MIAT knockout (KO) mice were performed to identify novel MIAT targets in the heart.

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Chromatin accessibility analysis of MIAT sufficient and MIAT deficient Th17 cells by ATAC-seq

Project description:T helper 17 (Th17) cells protect against fungal and bacterial infections and are implicated in autoimmunity. Several long intergenic noncoding RNAs (lincRNA) are induced during Th17 differentiation, however, their contribution to Th17 differentiation is poorly understood. We discovered a lincRNA myocardial infarction associated transcript (MIAT) to be upregulated early after induction of human Th17 cell differentiation along with an increase in the chromatin accessibility at the gene locus. We found that MIAT deficiency leads to chromatin accessibility at several loci including IL17A promoter.

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lncRNA and mRNA expression of cytokine treated MDA-MB-231 cells

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Long Intergenic Noncoding RNA MIAT Contributes to Human Th17 Cell Differentiation

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