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KDM4B regulates alternative splicing in castration resistant prostate cancer

ABSTRACT: We studied the alternative splicing function of KDM4B and found that KDM4B can act as a bona fide trans-acting splicing factor that binds RNA and signal-responsive scaffold protein that binds spliceosome.

ORGANISM(S): Homo sapiens

PROVIDER: GSE130620 | GEO | 2020/04/01

REPOSITORIES: GEO

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KDM4B regulates alternative splicing in castration resistant prostate cancer

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