Transcriptomics

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BCR-ABL1 promotes leukemia by converting p27 into a cytoplasmic oncoprotein


ABSTRACT: Coordinated BCR-ABL1 kinase-dependent and -independent mechanisms convert p27 from a nuclear tumor suppressor to a cytoplasmic oncogene. Persistence of oncogenic p27 functions despite effective inhibition of BCR-ABL1 may contribute to resistance to tyrosine kinase inhibitors.

ORGANISM(S): Mus musculus

PROVIDER: GSE59168 | GEO | 2014/10/08

SECONDARY ACCESSION(S): PRJNA254523

REPOSITORIES: GEO

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