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Emc1_Marquez - Disrupted ER membrane protein complex-mediated topogenesis drives congenital neural crest defects

ABSTRACT: Experiment consisted of MO knockdown of emc1 in Xenopus and comparison of proteome in emc1 depleted stage 24 embryos vs MO control injected embryos.

INSTRUMENT(S): Q Exactive

ORGANISM(S): Xenopus Tropicalis

TISSUE(S): Whole Body, Early Embryonic Cell

DISEASE(S): Congenital Heart Disease

SUBMITTER: Jonathan Marquez

LAB HEAD: Mustafa K Khokha

PROVIDER: PXD012770 | Pride | 2020-01-09

REPOSITORIES: Pride

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Dataset's files

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			Action	DRS
	LF18-020_Marquez_Khokha_rnk10_iso2_F508894_PRIDE_export.MSMS.mgf	Mgf
	LF18-020_Marquez_Khokha_rnk10_iso2_F508894_PRIDE_export.MSMS.pride.mgf.gz	Mgf
	LF18-020_Marquez_Khokha_rnk10_iso2_F508894_PRIDE_export.mzid.gz	Mzid
	LF18-020_Marquez_Khokha_rnk10_iso2_F508894_PRIDE_export.pride.mztab.gz	Mztab
	QEp18-1014_Khokha_Control_1.raw	Raw

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Disrupted ER membrane protein complex-mediated topogenesis drives congenital neural crest defects.

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