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Modulation of apple leaf trancriptome by Acibenzolar-S-methyl (ASM)

ABSTRACT: Modulation of apple leaf trancriptome by Acibenzolar-S-methyl (ASM)

PROVIDER: PRJNA1092251 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR28464957_1.fastq.gz	Fastqsanger.gz
	SRR28464957_2.fastq.gz	Fastqsanger.gz
	SRR28464958_1.fastq.gz	Fastqsanger.gz
	SRR28464958_2.fastq.gz	Fastqsanger.gz
	SRR28464959_1.fastq.gz	Fastqsanger.gz

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Similar Datasets

Modulation of apple leaf trancriptome by Acibenzolar-S-methyl (ASM)

Project description:Investigation of whole genome gene expression level changes in leaves of apple seedlings (Golden delicious) 3 days after treatment by Bion 50WG (ASM) versus water.

2024-04-01 | GSE262538 | GEO

Large scale analysis of leaf gene expression upon treatment of apple seedlings with acibenzolar-S-methyl or water

Project description:Investigation of whole genome gene expression level changes in the youngest expanded leaves of open-pollinated Golden Delicious seedlings either sprayed with reverse osmosis water or acibenzolar-S-methyl (ASM). Treatment was performed by spraying sprayed to runoff (with a pressurized hand sprayer) with the commercial product Bion 50 WG (Syngenta, Basel, Switzerland; 50% of ASM) prepared in reverse osmosis water at a final concentration of 0.4 g/L. The youngest developped leaf of each seedling was sampled 3 days after the treatment.

2021-06-11 | GSE176574 | GEO

Transcriptomic data from Malus domestica young leaves response to Acibenzolar-S-Methyl (ASM) treatment and nitrogen limitation or resupply.

Project description:Plant resistance inducers (PRIs) and nitrogen (N) nutrition are both factors known to act on plant defence but their interaction has been little studied and needs further research. The objective of the study was to further characterize this interaction on apple (Malus domestica) plantlets at the molecular level. The produced dataset allows the investigation of the molecular mechanisms underlying: i) the biotic resistance induced by ASM treatments on apple seedlings, ii) their response to different nitrogen (N) status (N limitation or subsequent N resupply), iii) the combination of both N status and ASM treatment.

2024-07-02 | GSE264541 | GEO

Large scale analysis of leaf gene expression upon treatment of apple grafted scions with acibenzolar-S-methyl or water at 20 or 35°C

Project description:Investigation of whole genome gene expression level changes in the youngest expanded leaves of GDDH13 grafted scions either sprayed with reverse osmosis water or acibenzolar-S-methyl (ASM) either exposed to control temperature (21°C/day ; 18°C/night) or three successive heat days (35°C for 4 h during the day ; 18°C/night). The morning of the third heat day, treatment was performed by spraying to runoff (with a pressurized hand sprayer) with the commercial product Bion 50 WG (Syngenta, Basel, Switzerland; 50% of ASM) prepared in reverse osmosis water at a final concentration of 0.4 g/L. All plants were then returned to control thermal conditions (21°C/day ; 18°C/night) for 2 days. The youngest developped leaf of each plant was sampled 72 hours after the treatment.

2024-10-31 | GSE280234 | GEO

Transcriptomic data from Malus domestica young leaves response to Acibenzolar-S-Methyl (ASM) treatment and nitrogen limitation or resupply.

Project description:Transcriptomic data from Malus domestica young leaves response to Acibenzolar-S-Methyl (ASM) treatment and nitrogen limitation or resupply.

| PRJNA1103119 | ENA

Comparative transcriptome analysis of rice roots treated with the resistance inducers piperonylic acid, acibenzolar-S-methyl and beta-aminobutyric acid

Project description:We used mRNA sequencing to study the effects of foliar treatment with piperonylic acid, acibenzolar-S-methyl or beta-aminobutyric acid on the root transcriptome of rice plants (cv. Kitaake) in order to find conserved systemic responses to distinct resistance-inducing stimuli

2022-12-06 | GSE194400 | GEO

Large scale analysis of leaf gene expression upon treatment of apple seedlings with acibenzolar-S-methyl or water

Project description:Large scale analysis of leaf gene expression upon treatment of apple seedlings with acibenzolar-S-methyl or water

| PRJNA736728 | ENA

Mechanisms and disease associations of haplotype-dependent allele specific DNA methylation: Methyl-seq data for the identification of hap-ASM

Project description:Haplotype-dependent allele-specific methylation (hap-ASM) can impact disease susceptibility, but maps of this phenomenon using stringent criteria in disease-relevant tissues remain sparse. Here we apply array-based and Methyl-seq approaches to multiple human tissues and cell types, including brain, purified neurons and glia, T lymphocytes, and placenta, and identify 795 hap-ASM differentially methylated regions (DMRs) and 3,082 strong methylation quantitative trait loci (mQTLs), most not previously reported. More than half of these DMRs have cell type-restricted ASM, and among them are 188 hap-ASM DMRs and 933 mQTLs located near GWAS signals for immune and neurological disorders. Targeted bis-seq confirmed hap-ASM in 12/13 loci tested, including CCDC155, CD69, FRMD1, IRF1, KBTBD11, and S100A*-ILF2, associated with immune phenotypes, MYT1L, PTPRN2,CMTM8 and CELF2, associated with neurological disorders, NGFR and HLA-DRB6, associated with both immunological and brain disorders, and ZFP57, a trans-acting regulator of genomic imprinting. Polymorphic CTCF and transcription factor (TF) binding sites are over-represented among hap-ASM DMRs and mQTLs, and analysis of the human data, supplemented by cross-species comparisons to Macaca mulattamacaques, indicates that CTCF and TF binding likelihood predicts the strength and direction of the allelic methylation asymmetry. These results show that hap-ASM is highly tissue-specific; an important trans-acting regulator of genomic imprinting is regulated by this phenomenon; variation in CTCF and TF binding sites is an underlying mechanism in primary tissues, and maps of hap-ASM and mQTLs reveal regulatory sequences underlying supera- and sub-threshold GWAS peaks in immunological and neurological disorders.

2016-03-21 | GSE79148 | GEO

Modulation of apple leaf trancriptome by tomato cutin monomer extract

Project description:Modulation of apple leaf trancriptome by tomato cutin monomer extract

| PRJNA1028998 | ENA

Acibenzolar-S-Methyl Reprograms Apple Transcriptome Toward Resistance to Rosy Apple Aphid.

Project description:Acibenzolar-S-methyl (ASM) is a chemical compound, which is able to induce resistance in several model and non-model plants, but the end-players of this induced defense remain ill-defined. Here, we test the hypothesis that treatment with ASM can protect apple (Malus × domestica) against the rosy apple aphid (Dysaphis plantaginea) and investigate the defense molecules potentially involved in resistance. We measured aphid life traits and performed behavioral assays to study the effect of ASM on plant resistance against the aphid, and then combined transcriptomic, bioinformatics, metabolic and biochemical analyses to identify the plant compounds involved in resistance. Plants treated with ASM negatively affected several life traits of the aphid and modified its feeding and host seeking behaviors. ASM treatment elicited up-regulation of terpene synthase genes in apple and led to the emission of (E,E)-?-farnesene, a sesquiterpene that was repellent to the aphid. Several genes encoding amaranthin-like lectins were also strongly up-regulated upon treatment and the corresponding proteins accumulated in leaves, petioles and stems. Our results link the production of specific apple proteins and metabolites to the antibiosis and antixenosis effects observed against Dysaphis plantaginea, providing insight into the mechanisms underlying ASM-induced herbivore resistance.

| S-EPMC6299034 | biostudies-literature

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