Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

ABSTRACT: Oral microbiome and periodontitis: a prospective study in postmenopausal women

PROVIDER: PRJNA796273 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
	SRR17550000_1.fastq.gz	Fastqsanger.gz
	SRR17550000_2.fastq.gz	Fastqsanger.gz
	SRR17550100_1.fastq.gz	Fastqsanger.gz
	SRR17550100_2.fastq.gz	Fastqsanger.gz
	SRR17550200_1.fastq.gz	Fastqsanger.gz

Items per page:

1 - 5 of 2410

Similar Datasets

Project description:Oral microbiome in pregnant women with periodontitis

| PRJNA1012892 | ENA

Phenotypic changes in PMN in Periodontits patients

Project description:The goal of this study is to use a rapid method for oral neutrophil isolation and use a transcriptomics approach to characterize and compare the neutrophil gene expression profile in the blood and oral compartment of healthy individuals, chronic periodontitis patients and refractory periodontitis patients. Total RNA obtained from isolated neutrophils from blood and oral samples of Healthy patients, chronic periodontits patients and refractory periodontitis patients

2013-02-26 | E-GEOD-43525 | biostudies-arrayexpress

Project description:The oral microbiome in young women at different stages of periodontitis

| PRJNA893254 | ENA

Project description:Oral microbiome in periodontitis

| PRJNA932553 | ENA

Project description:Oral microbiome dogs periodontitis

| PRJNA1099262 | ENA

Expression data from breast samples of postmenopausal women

Project description:The objective of the study was to comprehensively compare the genomic profiles in the breast of parous and nulliparous postmenopausal women to identify genes that permanently change their expression following pregnancy. The study was designed as a two-phase approach. In the discovery phase, we compared breast genomic profiles of 37 parous with 18 nulliparous postmenopausal women. In the validation phase, confirmation of the genomic patterns observed in the discovery phase was sought in an independent set of 30 parous and 22 nulliparous postmenopausal women.

2011-06-17 | E-GEOD-30010 | biostudies-arrayexpress

The Gut Microbiome Among Postmenopausal Latvian Women: The Influence of Dietary Habits

Project description:Gut microbiome in postmenopausal women, yoghurt experiment

| PRJEB52634 | ENA

Phenotypic changes in PMN in Periodontits patients

2013-02-26 | GSE43525 | GEO

Whole Sequencing Of The Mitochondrial Genome Of Breast Tumor Tissue In Mexican-Mestizo Postmenopausal Women, With Different Body Mass Index

Project description:Mitochondrial and oxidative stress have been related to obesity and breast cancer, and this cancer is more frequent and more aggressive in postmenopausal women with obesity. To investigate if Mexican-Mestizo postmenopausal women with breast cancer and obesity presented different somatic mutations in the mitochondrial DNA (mtDNA) when compared to women with normal body mass index.

2019-03-31 | GSE125801 | GEO

Epithelial-derived IL-23 promotes oral mucosal immunopathology

Project description:At mucosal surfaces, epithelial cells provide a structural barrier and an immune defense system. However, dysregulated epithelial responses can contribute to disease states. Here, we demonstrate that epithelial intrinsic production of IL-23 triggers an inflammatory loop in the prevalent oral disease, periodontitis. Epithelial IL-23 expression localized to areas proximal to the disease-associated microbiome, is evident both in experimental models and in patients with common and genetic forms of disease. Mechanistically, flagellated microbial species of the periodontitis microbiome, trigger epithelial IL-23 induction in a TLR5-dependent manner. Intriguingly, unlike other Th17-driven diseases, here non-hematopoietic cell-derived IL-23 serves as an initiator of pathogenic inflammation. Beyond periodontitis, analysis of publicly available datasets reveals expression of epithelial IL-23 in settings of infection, malignancy, and autoimmunity, suggesting a broader role for epithelial-intrinsic IL-23 in human disease. Collectively, this work highlights an unappreciated role for the barrier epithelium in the induction of IL-23-mediated inflammation.

2024-05-08 | GSE243173 | GEO

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

Tweets

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data