Cancer tolerance to chromosomal instability is driven by Stat1 inactivation in vivo
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ABSTRACT: Chromosomal instability (CIN) is a known hallmark of cancer, yet it also imposes stresses that reduce cellular fitness. In order to bridge the gap in knowledge on how cancer cells adapt to CIN-imposed stresses, we performed genome-wide transposon mutagenesis screen. Samples deposited in this database were acquired from mice used in our in vivo mutagenesis screen.
INSTRUMENT(S): NextSeq 500
ORGANISM(S): Mus musculus
SUBMITTER: René Wardenaar
PROVIDER: E-MTAB-11247 | biostudies-arrayexpress |
REPOSITORIES: biostudies-arrayexpress
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