Transcriptional response to stress in serum deprived mouse fibroblasts in the presence of MSK1/2 inhibitor.
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ABSTRACT: We have employed gene expression profiling in order to identify targets of transcriptional response to stress in resting mouse Swiss 3T3 fibroblasts, either untreated (control) or treated with anisomycin for 3 or 6 hours to induce the p38/MAP kinase pathway. In order determine transcriptional effects dependent on MSK1/2 kinase activity, H89 inhibitor was used in the study.
ORGANISM(S): Mus musculus
PROVIDER: GSE58746 | GEO | 2014/08/12
SECONDARY ACCESSION(S): PRJNA253332
REPOSITORIES: GEO
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